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Notch1 functions as a tumor suppressor in a model of K-ras-induced pancreatic ductal adenocarcinoma

Academic Article
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Overview

authors

  • Hanlon, L.
  • Avila, J. L.
  • Demarest, R. M.
  • Troutman, S.
  • Allen, M.
  • Ratti, F.
  • Rustgi, A. K.
  • Stanger, B.
  • Radtke, F.
  • Adsay, V.
  • Long, F.
  • Capobianco, A. J.
  • Kissil, Joseph

publication date

  • June 2010

journal

  • Cancer Research  Journal

subject areas

  • Animals
  • Carcinoma, Pancreatic Ductal
  • Disease Progression
  • Gene Deletion
  • Gene Expression Regulation, Neoplastic
  • Genes, Tumor Suppressor
  • Genes, ras
  • Mice
  • Mice, Transgenic
  • Pancreatic Neoplasms
  • Receptor, Notch1
  • Signal Transduction
  • beta Catenin
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Identity

PubMed Central ID

  • PMC2880196

International Standard Serial Number (ISSN)

  • 0008-5472

Digital Object Identifier (DOI)

  • 10.1158/0008-5472.can-09-4645

PubMed ID

  • 20484026
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Additional Document Info

start page

  • 4280

end page

  • 4286

volume

  • 70

issue

  • 11

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