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Fatty acid amide hydrolase and monoacylglycerol lipase inhibitors produce anti-allodynic effects in mice through distinct cannabinoid receptor mechanisms

Academic Article
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Overview

related to degree

  • Long, Jonathan, Ph.D. in Chemistry, Scripps Research 2007 - 2011

authors

  • Kinsey, S. G.
  • Long, Jonathan
  • Cravatt, Benjamin
  • Lichtman, A. H.

publication date

  • December 2010

journal

  • Journal of Pain  Journal

subject areas

  • Amidohydrolases
  • Animals
  • Enzyme Inhibitors
  • Female
  • Male
  • Mice
  • Mice, Knockout
  • Monoacylglycerol Lipases
  • Neuralgia
  • Receptor, Cannabinoid, CB1
  • Receptor, Cannabinoid, CB2
  • Sciatic Nerve
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Research

keywords

  • 2-ararchidonylglycerol (2-AG)
  • Endogenous cannabinoid
  • anandamide
  • fatty acid amide hydrolase (FAAH)
  • monoacylglycerol lipase (MAGL)
  • neuropathic pain
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Identity

PubMed Central ID

  • PMC2962430

International Standard Serial Number (ISSN)

  • 1526-5900

Digital Object Identifier (DOI)

  • 10.1016/j.jpain.2010.04.001

PubMed ID

  • 20554481
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Additional Document Info

start page

  • 1420

end page

  • 1428

volume

  • 11

issue

  • 12

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