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Increasing the receptor tyrosine kinase EphB2 prevents amyloid-β-induced depletion of cell surface glutamate receptors by a mechanism that requires the PDZ-binding motif of EphB2 and neuronal activity

Academic Article
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Overview

authors

  • Miyamoto, Takashi
  • Kim, D.
  • Knox, J. A.
  • Johnson, E.
  • Mucke, L.

publication date

  • 2016

journal

  • Journal of Biological Chemistry  Journal

subject areas

  • Alzheimer Disease
  • Amino Acid Motifs
  • Amyloid beta-Peptides
  • Animals
  • Cells, Cultured
  • Female
  • Hippocampus
  • Humans
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • Nerve Tissue Proteins
  • Neurons
  • PDZ Domains
  • Protein Binding
  • Receptor, EphB2
  • Receptors, AMPA
  • Receptors, N-Methyl-D-Aspartate
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Identity

PubMed Central ID

  • PMC4722453

International Standard Serial Number (ISSN)

  • 0021-9258

Digital Object Identifier (DOI)

  • 10.1074/jbc.M115.666529

PubMed ID

  • 26589795
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Additional Document Info

start page

  • 1719

end page

  • 1734

volume

  • 291

issue

  • 4

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