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Loss of sorting nexin 27 contributes to excitatory synaptic dysfunction by modulating glutamate receptor recycling in Down's syndrome

Academic Article
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  • Identity
  • Additional Document Info
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Overview

authors

  • Wang, X.
  • Zhao, Y.
  • Zhang, X.
  • Badie, H.
  • Zhou, Y.
  • Mu, Y.
  • Loo, L. S.
  • Cai, L.
  • Thompson, R. C.
  • Yang, B.
  • Chen, Y.
  • Johnson, P. F.
  • Wu, C.
  • Bu, G.
  • Mobley, W. C.
  • Zhang, D.
  • Gage, F. H.
  • Ranscht, B.
  • Zhang, Y. W.
  • Lipton, Stuart
  • Hong, W.
  • Xu, H.

publication date

  • April 2013

journal

  • Nature Medicine  Journal

subject areas

  • Animals
  • Antineoplastic Combined Chemotherapy Protocols
  • C-Reactive Protein
  • CCAAT-Enhancer-Binding Protein-beta
  • CCAAT-Enhancer-Binding Proteins
  • Cerebral Cortex
  • Cisplatin
  • Down Syndrome
  • Down-Regulation
  • Female
  • Hippocampus
  • Humans
  • Ifosfamide
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Knockout
  • MicroRNAs
  • Mitomycin
  • Nerve Tissue Proteins
  • Receptors, Glutamate
  • Sorting Nexins
  • Synapses
  • Transcription Factors
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Identity

PubMed Central ID

  • PMC3911880

International Standard Serial Number (ISSN)

  • 1078-8956

Digital Object Identifier (DOI)

  • 10.1038/nm.3117

PubMed ID

  • 23524343
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Additional Document Info

start page

  • 473

end page

  • 480

volume

  • 19

issue

  • 4

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