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PPAR alpha

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  • 5-aminoimidazole-4-carboxyamide-ribonucleoside (AICAR)-stimulated hepatic expression of Cyp4a10, Cyp4a14, Cyp4a31, and other peroxisome proliferator-activated receptor alpha-responsive mouse genes is AICAR 5'-monophosphate-dependent and AMP-activated protein kinase-independent  Academic Article
  • Antagonizing peroxisome proliferator-activated receptor α activity selectively enhances Th1 immunity in male mice  Academic Article
  • Effect of muraglitazar on death and major adverse cardiovascular events in patients with type 2 diabetes mellitus  Academic Article
  • Effects of the endogenous PPAR-alpha agonist, oleoylethanolamide on MDMA-induced cognitive deficits in mice  Academic Article
  • Genomic profiles and predictors of early allograft dysfunction after human liver transplantation  Academic Article
  • Identification of the first potent, selective and bioavailable PPARα antagonist  Academic Article
  • Interference with acute nausea and anticipatory nausea in rats by fatty acid amide hydrolase (FAAH) inhibition through a PPARα and CB1 receptor mechanism, respectively: a double dissociation  Academic Article
  • KLF5 Is Induced by FOXO1 and Causes Oxidative Stress and Diabetic Cardiomyopathy  Academic Article
  • Lack of sterol regulatory element binding factor-1c imposes glial Fatty Acid utilization leading to peripheral neuropathy  Academic Article
  • Nuclear receptor/microRNA circuitry links muscle fiber type to energy metabolism  Academic Article
  • Opposing roles of peroxisome proliferator-activated receptor alpha and growth hormone in the regulation of CYP4A11 expression in a transgenic mouse model  Academic Article
  • PPARG post-translational modifications regulate bone formation and bone resorption  Academic Article
  • Proteomics analysis of cardiac muscle from rats with peroxisomal proliferator-activated receptor α (PPARα) stimulation  Academic Article
  • Regulation of carbohydrate metabolism by the farnesoid x receptor  Academic Article
  • β-Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR-α, PPAR-γ and TRPV1, but not CB₁ or CB₂ receptors  Academic Article

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