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Common charge-shift mutation glu65lys in k(+) channel beta(1)-subunit kcnmb1: Pleiotropic consequences for glomerular filtration rate and progressive renal disease

Academic Article
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  • Research
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Overview

authors

  • Chen, Y. Q.
  • Salem, R. M.
  • Rao, F. W.
  • Fung, M. M.
  • Bhatnagar, V.
  • Pandey, B.
  • Mahata, M.
  • Waalen, Jill
  • Nievergelt, C. M.
  • Lipkowitz, M. S.
  • Hamilton, B. A.
  • Mahata, S. K.
  • O'Connor, D. T.

publication date

  • 2010

journal

  • American Journal of Nephrology  Journal

subject areas

  • African Americans
  • Aged
  • Alleles
  • Cohort Studies
  • Disease Progression
  • European Continental Ancestry Group
  • Female
  • Gene Frequency
  • Glomerular Filtration Rate
  • Heterozygote
  • Humans
  • Hypertension
  • Large-Conductance Calcium-Activated Potassium Channel beta Subunits
  • Linear Models
  • Male
  • Middle Aged
  • Mutation
  • Nephrosclerosis
  • Phenotype
  • Renal Insufficiency, Chronic
  • Reproducibility of Results
  • Sensitivity and Specificity
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Research

keywords

  • African-American Study of Kidney Disease and Hypertension
  • Glomerular filtration rate
  • Hypertensive nephrosclerosis
  • KCNMB1
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Identity

PubMed Central ID

  • PMC2975731

International Standard Serial Number (ISSN)

  • 0250-8095

Digital Object Identifier (DOI)

  • 10.1159/000320131

PubMed ID

  • 20861615
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Additional Document Info

start page

  • 414

end page

  • 424

volume

  • 32

issue

  • 5

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