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Control of experimental spasticity by targeting the degradation of endocannabinoids using selective fatty acid amide hydrolase inhibitors

Academic Article
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Overview

related to degree

  • Long, Jonathan, Ph.D. in Chemistry, Scripps Research 2007 - 2011

authors

  • Pryce, G.
  • Cabranes, A.
  • Fernandez-Ruiz, J.
  • Bisogno, T.
  • Di Marzo, V.
  • Long, Jonathan
  • Cravatt, Benjamin
  • Giovannoni, G.
  • Baker, D.

publication date

  • December 2013

journal

  • Multiple Sclerosis Journal  Journal

subject areas

  • Amidohydrolases
  • Animals
  • Arachidonic Acids
  • Brain
  • Disease Models, Animal
  • Encephalomyelitis, Autoimmune, Experimental
  • Endocannabinoids
  • Enzyme Inhibitors
  • Female
  • Male
  • Mice
  • Mice, Biozzi
  • Mice, Knockout
  • Molecular Targeted Therapy
  • Monoacylglycerol Lipases
  • Muscle Spasticity
  • Muscle, Skeletal
  • Polyunsaturated Alkamides
  • Time Factors
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Research

keywords

  • Anandamide
  • endocannabinoid
  • experimental autoimmune encephalomyelitis
  • fatty acid amide hydrolase
  • multiple sclerosis
  • spasticity
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Identity

International Standard Serial Number (ISSN)

  • 1352-4585

Digital Object Identifier (DOI)

  • 10.1177/1352458513485982

PubMed ID

  • 23625705
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Additional Document Info

start page

  • 1896

end page

  • 1904

volume

  • 19

issue

  • 14

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