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Pten mutations alter brain growth trajectory and allocation of cell types through elevated β-catenin signaling

Academic Article
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Overview

related to degree

  • Huang, Wen-Chin, Ph.D. in Biology, Scripps Research 2012 - 2017

authors

  • Chen, Y.
  • Huang, Wen-Chin
  • Sejourne, J.
  • Clipperton-Allen, A. E.
  • Page, Damon

publication date

  • July 2015

journal

  • Journal of Neuroscience  Journal

subject areas

  • Animals
  • Animals, Newborn
  • Brain
  • Carrier Proteins
  • Cell Cycle
  • Cell Cycle Proteins
  • Embryo, Mammalian
  • Female
  • Gene Expression Regulation
  • Haploinsufficiency
  • Homeodomain Proteins
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Nerve Tissue Proteins
  • Neuroglia
  • Neurons
  • PTEN Phosphohydrolase
  • Signal Transduction
  • TOR Serine-Threonine Kinases
  • Transcription Factors
  • beta Catenin
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Research

keywords

  • Pten
  • autism
  • beta-catenin
  • brain growth
  • cell types
  • macrocephaly
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Identity

International Standard Serial Number (ISSN)

  • 0270-6474

Digital Object Identifier (DOI)

  • 10.1523/jneurosci.5272-14.2015

PubMed ID

  • 26180201
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Additional Document Info

start page

  • 10252

end page

  • 10267

volume

  • 35

issue

  • 28

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