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DPP9 enzyme activity controls survival of mouse migratory tongue muscle progenitors and its absence leads to neonatal lethality due to suckling defect

Academic Article
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Overview

authors

  • Kim, M.
  • Minoux, M.
  • Piaia, A.
  • Kueng, B.
  • Gapp, B.
  • Weber, D.
  • Haller, C.
  • Barbieri, S.
  • Namoto, Kenji
  • Lorenz, T.
  • Wirsching, J.
  • Bassilana, F.
  • Dietrich, W.
  • Rijli, F. M.
  • Ksiazek, I.

publication date

  • November 2017

journal

  • Developmental Biology  Journal

subject areas

  • Alanine
  • Animals
  • Animals, Newborn
  • Animals, Suckling
  • Catalytic Domain
  • Cell Count
  • Cell Survival
  • Dipeptidyl-Peptidases and Tripeptidyl-Peptidases
  • Mice
  • Mice, Transgenic
  • Muscle Development
  • Muscle Proteins
  • Muscle, Skeletal
  • Point Mutation
  • Receptors, CXCR4
  • Serine
  • Stem Cells
  • Tongue
  • Tongue Diseases
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Research

keywords

  • DPP9
  • Microglossia
  • Muscle progenitors
  • Neonatal lethality
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Identity

International Standard Serial Number (ISSN)

  • 0012-1606

Digital Object Identifier (DOI)

  • 10.1016/j.ydbio.2017.09.001

PubMed ID

  • 28887018
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Additional Document Info

start page

  • 297

end page

  • 308

volume

  • 431

issue

  • 2

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