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Dominant-interfering forms of MEF2 generated by caspase cleavage contribute to NMDA-induced neuronal apoptosis

Academic Article
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  • Identity
  • Additional Document Info
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Overview

authors

  • Okamoto, S.
  • Li, Z.
  • Ju, C.
  • Scholzke, M. N.
  • Mathews, E.
  • Cui, J.
  • Salvesen, G. S.
  • Bossy-Wetzel, E.
  • Lipton, Stuart

publication date

  • March 2002

journal

  • Proceedings of the National Academy of Sciences of the United States of America  Journal

subject areas

  • Animals
  • Apoptosis
  • Caspase 3
  • Caspase 7
  • Caspases
  • Cells, Cultured
  • Cerebral Cortex
  • DNA
  • DNA-Binding Proteins
  • Enzyme Activation
  • Gene Expression Regulation
  • MEF2 Transcription Factors
  • Mice
  • Mice, Inbred C57BL
  • Mutation
  • Myogenic Regulatory Factors
  • N-Methylaspartate
  • Neurons
  • Protein Binding
  • Protein Processing, Post-Translational
  • RNA, Messenger
  • Rats
  • Reperfusion Injury
  • Response Elements
  • Stroke
  • Transcription Factors
  • Transcription, Genetic
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Identity

PubMed Central ID

  • PMC122633

International Standard Serial Number (ISSN)

  • 0027-8424

Digital Object Identifier (DOI)

  • 10.1073/pnas.022036399

PubMed ID

  • 11904443
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Additional Document Info

start page

  • 3974

end page

  • 3979

volume

  • 99

issue

  • 6

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