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Ginsenoside Re rescues methamphetamine-induced oxidative damage, mitochondrial dysfunction, microglial activation, and dopaminergic degeneration by inhibiting the protein kinase Cδ gene

Academic Article
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Overview

authors

  • Shin, E. J.
  • Shin, S. W.
  • Nguyen, T. T. L.
  • Park, D. H.
  • Wie, M. B.
  • Jang, Choon-Gon
  • Nah, S. Y.
  • Yang, B. W.
  • Ko, S. K.
  • Nabeshima, T.
  • Kim, H. C.

publication date

  • June 2014

journal

  • Molecular Neurobiology  Journal

subject areas

  • Animals
  • Dopaminergic Neurons
  • Ginsenosides
  • Methamphetamine
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Microglia
  • Mitochondria
  • Nerve Degeneration
  • Oxidative Stress
  • Protein Kinase C-delta
  • Protein Kinase Inhibitors
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Research

keywords

  • Apoptosis
  • Dopamine
  • Ginsenoside Re
  • Methamphetamine
  • Mitochondria
  • Oxidative damage
  • Protein kinase C delta
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Identity

International Standard Serial Number (ISSN)

  • 0893-7648

Digital Object Identifier (DOI)

  • 10.1007/s12035-013-8617-1

PubMed ID

  • 24430743
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Additional Document Info

start page

  • 1400

end page

  • 1421

volume

  • 49

issue

  • 3

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