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The CAMKK2-AMPK kinase pathway mediates the synaptotoxic effects of Aβ oligomers through Tau phosphorylation

Academic Article
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Overview

authors

  • Mairet-Coello, G.
  • Courchet, J.
  • Pieraut, S.
  • Courchet, V.
  • Maximov, Anton
  • Polleux, Franck

publication date

  • April 2013

journal

  • Neuron  Journal

subject areas

  • Action Potentials
  • Amyloid beta-Peptides
  • Amyloid beta-Protein Precursor
  • Animals
  • Brain
  • Calcium
  • Calcium-Calmodulin-Dependent Protein Kinase Kinase
  • Cells, Cultured
  • Dendritic Spines
  • Dose-Response Relationship, Drug
  • Electroporation
  • Embryo, Mammalian
  • Enzyme Inhibitors
  • Gene Expression Regulation, Enzymologic
  • Glutamic Acid
  • Green Fluorescent Proteins
  • Humans
  • Mice
  • Mice, Inbred C57BL
  • Mice, Transgenic
  • Mutation
  • Neurons
  • Patch-Clamp Techniques
  • Peptide Fragments
  • Phosphorylation
  • Platelet-Derived Growth Factor
  • Protein Kinases
  • Serine
  • Transfection
  • tau Proteins
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Identity

PubMed Central ID

  • PMC3784324

International Standard Serial Number (ISSN)

  • 1097-4199 (Electronic) 0896-6273 (Linking)

Digital Object Identifier (DOI)

  • 10.1016/j.neuron.2013.02.003

PubMed ID

  • 23583109
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Additional Document Info

start page

  • 94

end page

  • 108

volume

  • 78

issue

  • 1

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